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Genetic and epigenic association studies have produced inconsistent results in BD, especially with regards to the Val66Met BDNF polymorphism which generated initial transdiagnostic interest, but has since failed to garner consistent associations across subpopulations in BD [161]

Several studies have noted elevated activity in other limbic and subcortical areas, including insula, ventral striatum, putamen, hypothalamus, and thalamus, in bipolar depression relative to healthy controls (106, 137, 139)

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It is worth noting that B OTS964 differs from intermediates previously reported during spliceosome activation 12,97,98,99 , in which both NTC and NTR complexes are either bound 12,97 or absent 98,99