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Some are pathogenic themselves and cause their own set of symptoms, while others act alongside Borrelia burgdorferi to create a biofilm that makes them resistant to antibiotics

The documented mechanisms are: VEGF stimulation in dermal fibroblasts that supports the microvascular network feeding the follicle, collagen and glycosaminoglycan synthesis at the follicular basement membrane, anti-inflammatory signaling that calms the chronic micro-inflammation associated with androgenetic alopecia, and direct follicular signaling that extends the anagen growth phase

Once all of the medication goes in you can stop pinching the skin and take the needle out

GHK-Cu is being explored for its potential applications in skin regeneration, hair follicle support, wound healing, and inflammation control