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This section delineates SFNs multifaceted mechanisms across epigenetic regulation, programmed cell death pathways (particularly ferroptosis), and tumor immune microenvironment remodeling, establishing its role as a truly multi-targeted therapeutic agent

Intranasal delivery of MSC-EVs in TBI animal models has been shown to promote neurofunctional recovery, suppress NLRP3 inflammasome activation, inhibit p38 MAPK signalling, and prevent microglial polarization toward the pro-inflammatory M1 phenotype, collectively improving behavioural and cognitive outcomes [393]

Zhang M, Hu S, Liu L, et al