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Mechanism Involving Nrf2-Keap1 Complex Mitigates the NLRP3 Inflammasome in PD In PD, the NLRP3 inflammasome acts as a central driver of neuroinflammation, perpetuating a self-sustaining inflammatory loop that accelerates the degeneration of dopaminergic neurons

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This mechanism is largely route-independent at the cellular level, which is why it is invoked for both topical and injectable use but a shared mechanism does not by itself prove that either route produces a visible skin outcome

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