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Melatonin suppresses ILC2 function via NRF2 activation Whilst we found that melatonins immunosuppressive effects on ILC2s were glutathione dependent, the upstream mechanism linking melatonin to glutathione biosynthesis remained unclear

This type of study is important for several reasons: it tells us how long the peptide remains active in the body, what breakdown products are formed (which could themselves have biological activity or be used as markers in drug testing), and helps explain why AOD9604 needs to be administered in certain ways to remain effective

doi: 10.1016/j.wneu.2013.07.093

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