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Gliclazide acts by binding to the sulfonylurea receptors (SUR) situated on the cells of the pancreas and acts by closing the ATP-dependent potassium channels that lead to an influx of calcium ions and releasing insulin by process of exocytosis

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Such effects may result in impaired synaptic pruning and phagocytic clearance of excess synapses by immature microglia during postnatal brain development

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