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For CIDP, evidence is also emerging that patients previously maintained on IVIg treatment may undergo disease worsening upon switching to FcRn antagonists, underlining the fact that other disease drivers than solely auto-Abs are also vital aspects of pathophysiology in at least certain patients with CIDP [265, 401, 402]

showed that BMAL1 regulates transcriptional activity by binding to cis-regulatory elements within inflammatory gene promoters, thereby controlling their circadian expression (113)

(2020) 15:e0235929

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