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Notably, the dual role of EMFs manifests in their ability to induce pro-metastatic phenotypes under certain parameters [7,8] while exhibiting therapeutic potential under others [9-12] [Table 1]

These studies are designed to test the hypothesis that activating distinct, yet complementary, neurohormonal pathwaysthe hindbrain-centric amylin pathway and the forebrain/hypothalamic GLP-1 pathwaycan produce additive or synergistic effects on appetite suppression and weight reduction

These often resolve without any protocol changes

It has been shown to improve insulin sensitivity, reduce obesity, and prevent diet-induced insulin resistance