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In vitro data and mouse studies suggest that NE proteolytic activity triggers an inflammatory process that induces goblet cell metaplasia, which results in overexpression of mucins that contribute to the symptomatology of CRS (66, 67)

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The findings presente the promise of MF-based neuromodulation as a non-invasive, highly targeted therapeutic strategy for conditions such as epilepsy, movement disorders, and neurodegenerative diseases, with potential applications in chronic pain management and future clinical interventions