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However, both genome-wide association studies (GWASs) and candidate gene studies have confirmed an important role for human leukocyte antigen (HLA) class I and class II genes in some but not all forms of DILI

To test if this mechanism was at play upon depleting GSH in the liver, we treated Gclc L-KO mice with T0901317, an LXR agonist

The purpose of this review is to provide a state-of-the-art translational approach to arterial stiffness, highlighting unique pathophysiological mechanisms (e.g., perivascular adipose tissue (PVAT), extracellular vesicles), clinical relevance, and future directions

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