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In the shuttle, glutamate is converted to glutamine in astrocytes, and then exported to neuronal system A transporters, where it is converted to glutamate intracellularly by phosphate activated glutaminase (21)

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We developed separate SEM models for participants with and without NDR because previous studies suggest that mitochondrial function in ASD, especially in relation to handling oxidative stress, is different depending on the history of NDR [25, 26, 30, 31]

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