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These well-characterized findings show that immature red blood cells suffer ferroptosis stress from iron accumulation and oxygen during stress erythropoiesis, while ferroptosis is rarely detectable in normal red blood cells

Beyond serving as a reducing cofactor for antioxidant enzymes, GSH exerts direct antioxidant activity by scavenging reactive oxygen species (ROS) and RNS, including superoxide anions (O 2 ), H 2 O 2 , and hydroxyl radicals (OH) 3

It is natural to speculate whether the inhibition of GS and GCL is an important evolutionary mechanism to keep GSH high, but not too high, in females during the childbearing years

Here we identify an underappreciated role for GSH as a cysteine reservoir for tumours