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The above genome-wide studies did not detect any profound methylation of NeuroD1 and NeuroD2 in ESC-derived glutamatergic neurons, the brain tissue and various non-neural differentiated cells (Meissner et al., 2008

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Depending on dose, a fraction of APAP is converted into a highly reactive toxic intermediate, N-acetyl-p-benzoquinone imine (NAPQI) by several P450 cytochromes[4]

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