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Unlike natural IGF-1, IGF-1 LR3 was designed with an extended half-life and increased bioavailability, allowing for prolonged biological activity and enhanced therapeutic potential

When endogenous neurons are ablated through apoptosis, resulting in minimal reactive gliosis and inflammation, tNs can achieve near-perfect integration with appropriate brain-wide synaptic inputs and functional properties comparable to endogenous neurons (Falkner et al., 2016)