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Studies converge toward significant reduction of ulcer area and accelerated reepithelialization after systemic BPC-157 administration

These findings are very interesting, considering the essential role of NO in healing, and suggest the possibility of overriding of this negative effect and the neuroprotective capabilities of BPC-157

While intermediate metabolites associated with TCA cycle (Tricarboxylic Acid Cycle) such as fumarate, succinate, and malate were not significantly different between the groups, we suggest a decrease in mitochondrial activity may result in lower ATP generation as reported in other inflammatory conditions (65, 66)