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Loren Pickart, who found that it caused older human liver tissue to produce proteins more characteristic of younger tissue.2 3 This resulted in GHK being originally described, specifically, as a liver cell growth factor

BPC-157 modulates the VEGFR2 pathway to promote angiogenesis, activates the FAK-paxillin system to enhance cell migration toward injury sites, and influences nitric oxide production for improved blood flow

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Because the drivers are systemic, getting a succinct explanation is a challenge so I was grateful for the opportunity to hone the story and I got some excellent feedback