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Description

The FABP4-TLR4 signaling axis has consequently emerged as a promising therapeutic target for improving insulin sensitivity in T2DM

FSP1: ferroptosis suppressor protein 1

Most of the time, more than 95%, no surgery is needed

Transcription of NLRP3 and inflammatory factors is upregulated through the Toll-like receptor (TLR)-mediated NF-B pathway in response to various cellular stresses, including ROS, mitochondrial damage, and lysosomal destabilization, promoting inflammasome oligomerization and recruitment of other components (Coll et al., 2022)