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Excess androgens driven by insulin resistance blunt the progesterone and oestrogen feedback that normally regulates kisspeptin neuronal activity

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Researchers investigate BPC-157, a 15 amino acid peptide fragment, alongside the copper binding GHK tripeptide motif and TB-500, a thymosin beta 4 analogue fragment, in models involving connective tissue, angiogenesis signaling, and cellular migration pathways

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