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Previous studies revealed that TP exerted therapeutic effects in RA by targeting RA-associated proteins (such as NF-B, MMP-9 and JNK), accompanied by reduced activity of FLSs and Th17 differentiation (108110)

this is probably due to the high CMFDA fluorescence in the perinuclear area, which could be explained by the fact that BSO, at the concentrations used in this work, is unable to deplete mitochondrial GSH [34], [35]

GABAA-receptor heterogeneity in the adult rat brain: differential regional and cellular distribution of seven major subunits

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