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In ALS, the dysfunctional uptake of glutamate by astrocytic transporters contributes to excitotoxicity at least in a subset of ALS patients [97], however, simple inhibition of glutamate transport failed to produce an ALS-like condition in animals (see section The classical view: microglia as engines of inflammation for more details)

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Outside primary metabolic indications, these mechanisms are also of interest for cardiometabolic risk reduction (such as waist circumference, triglycerides, and blood pressure) and for appetite control during lifestyle-based weight loss plans