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These results suggest that BPC 157 may indirectly form part of the immune systems support

Fibrosis in Non-Alcoholic Steatohepatitis In addition to exacerbating hepatocyte injury and apoptosis, activation of Kupffer cells and expansion of the inflammatory niche stimulates differentiation of liver-resident HSCs into pro-fibrogenic myofibroblasts (192) ( Figure 1B )