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However, it may be because extracellular matrix (ECM) accumulation and apoptosis work together in several different pathogenic mechanisms to cause the injury and progression of kidney disease (34)

CPP-CAND disrupts FOXO4-p53 foci and induces apoptosis through caspase-dependent pathways FOXO4 is progressively recruited to euchromatin foci following senescence induction and is retained in the nucleus of senescent cells through its interaction with p53, contributing to the maintenance of the senescent state 34

Thus, it is likely that accelerated cellular aging is involved in the pathophysiology of anxiety