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Looking forward, the clinical application of ferroptosis-based therapies in PDAC hinges on overcoming several pivotal challenges

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ChREBP was down-regulated in patients with NAFLD ( n = 22) compared to healthy controls ( n = 10), and instead, SREBP1c was reported to be one of the predominant regulators of DNL in NAFLD, upregulating genes coding for ACC1 and FAS [53]