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Persistent mitochondrial OXPHOS in CRC cells enables metabolic compensation that undermines EMP-targeted therapies, necessitating combinatorial inhibition of complementary pathways (e.g., TCA cycle, PPP) for durable efficacy

Therefore, the biosynthesis, subcellular localization, and systemic transport of NAD and its intermediates are key to the regulation of various biological processes with significant impact on cellular and tissue functionality

Their further investigation revealed that in renal cancer cells, high expression of the hypoxia-inducible factor (HIF) pathways hypoxia-inducible factor 2 enhances the sensitivity of ccRCC to ferroptosis by enriching unsaturated fatty acids through hilpda

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